By Burton J. Bogitsh
ISBN-10: 012415915X
ISBN-13: 9780124159150
Human Parasitology emphasizes a scientific standpoint whereas incorporating useful morphology, body structure, biochemistry, and immunology to reinforce appreciation of the varied implications of parasitism. Bridging the distance among classical scientific parasitology texts and standard encyclopedic treatises, Human Parasitology appeals to scholars attracted to the clinical facets of parasitology and those that require an outstanding starting place within the biology of parasites. This 4e has been totally revised to combine the latest molecular discoveries approximately mosquitoes, ticks and different arthropods as vectors, as good because the most well known healing regimens.
New to this variation: . good points increased insurance of the evolution of parasitism and an in depth replace to immunology of parasite-host interactions . bargains an more suitable artwork software that includes life-cycle illustrations and extra SEM and TEM micrographs . New Host Immune reaction part for every organism
- Presents various parasites and examines how they have interaction with their hosts and reply to new treatments
- Easy to learn with photos, diagrams, and scientific case photos throughout
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Extra resources for Human Parasitology
Example text
Any breakdown in T cell function serves to upgrade the infection, often resulting in acute central nervous system involvement in immunocompromised individuals. However, T. cruzi does not appear to exacerbate the effects of dual infections with HIV as Leishmania does. Plasmodium spp. (see p. 116) The causative agent of human malaria displays two life cycle phases in its human host, an extraerythrocytic phase initiated when sporozoites infect hepatocytes and an erythocytic phase involving the infection of red blood cells by merozoites.
Histolytica cysts from those of E. coli, in which the chromatoidal bars have jagged or splintered ends. The nucleus undergoes two mitotic divisions to produce four vesicular nuclei in the mature cyst of E. histolytica (Fig. 4-1). Such cysts represent the infective form and pass out of the host in feces, after which the glycogen and chromatoidal substance are slowly metabolized and disappear. Cysts of E. , chlorinated compounds, fluorides). Cysts in water can survive for a month, while those in feces on dry land can survive for more than 12 days; they tolerate temperatures up to a thermal death point of 50 C.
IFNg). Macrophage activation is a major mechanism by which the body eliminates infectious organisms. IFN-g activates macrophages, which, in turn, produce effectors such as toxic oxygen radical intermediates, reactive nitrogen intermediates, TNF, and lysosomal enzymes. Such macrophage activity enables the host to either kill intracellular parasites or, in many instances, keep their numbers in check. Worldwide, parasitic infections account for a higher incidence of morbidity and mortality than diseases produced by any other group of organisms.
Human Parasitology by Burton J. Bogitsh
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